DOI : 10.1055/s-00000094

Zeitschrift für Gastroenterologie

Issue 01 · Volume 52 · January 2014 DOI: 10.1055/s-004-26267

30. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber

Tübingen, 24. – 25. 1. 2014
Kongresspräsident: Prof. Dr. Alfred Königsrainer


V_3_03
Sowa, JP; Wingerter, L; Gerken, G; Palmert, M; Canbay, A; Grasemann, C: Offspring of high fat diet fed dams develop hepatic steatosis
P_3_01
P_3_02
P_3_03
Hochrath, K; Hölter, SM; Garrett, L; Gailus-Durner, V; Fuchs, H; Hrabě de Angelis, M; Wurst, W; Stokes, CS; Lammert, F: ABCB4-defiziente Mäuse zeigen depressives und ängstliches Verhalten
P_3_04
Gehrke, N; Zinngrebe, H; Mann, A; Nagel, M; Alt, Y; Schad, A; Galle, PR; Wörns, MA; Schattenberg, JM: Bedeutung des Caspase 8-Homologs cFLIP bei der metabolischen Leberschädigung
P_3_05
Ahmad, S; Giannelli, V; Naz, N; Malik, I; Amanzada, A; Ramadori, G; Moriconi, F: Changes of gene expression of different BMP-proteins in liver in different mouse and rat models of acute phase reaction
P_3_06
Wönne, EC; Hiller, T; Przibilla, J; Müller-Vieira, U; Guthke, R; Schmidt-Heck, W; Damm, G; Knöspel, F; Richter, M; koczan, D; Nüssler, AK; Schulz, A; Zeilinger, K: Comparison of the response of primary rat and human hepatocytes to ethanol exposition in a miniaturized three-dimensional (3D) multi-compartment bioreactor system
P_3_08
Martius, G; Alwahsh, SM; Rave-Fränk, M; Hess, CF; Christiansen, H; Ramadori, G; Malik, IA: Effect of direct single-dose irradiation on fat metabolism in rat liver: FAT/CD36 as the main responsible protein?
P_3_11
Sperling, S; Herzog, N; Hansen, M; Küpper, JH; Seehofer, D; Damm, G: Genetically engineered hepatocytes as a capable application for in vitro liver cell systems
P_3_12
Freyer, N; Strahl, N; Knöspel, F; Urbaniak, T; Zeilinger, K: Hepatic differentiation of hiPSC in a 3D multicompartment bioreactor
P_3_13
Döring, S; Seeßle, J; Tuma, S; Stremmel, W; Chamulitrat, W: Hepatic FATP4-deficiency causes alterations in lipid metabolism
P_3_15
Levada, K; Güldiken, N; Pfaff, CM; Mo, FR; Lahiri, P; Trautwein, C; Strnad, P: Hsp72 overexpression protects from acetaminophen and DDC-associated liver injury but enhances protein aggregation
P_3_17
Thiel, C; Matz-Soja, M; Sales, S; Eibisch, M; Schiller, J; Shevchenko, A; Gebhardt, R: Influence of Carbon Chain Length on Formation and Composition of Triglycerides in Free Fatty Acid- induced Steatosis
P_3_18
Schließ, F; Hoehme, S; Henkel, SG; Ghallab, A; Driesch, D; Boettger, J; Guthke, R; Pfaff, M; Hengstler, JG; Gebhardt, R; Häussinger, D; Drasdo, D; Zellmer, S: Integrierte metabolische und räumlich-zeitliche Modellierung des Ammoniak-Stoffwechsels während der Leber-Schädigung und -Regeneration
P_3_19
Schreiter, T; Sowa, J; Mathe, Z; Treckmann, J; Broecker-Preuß, M; Baba, H; Gieseler, RK; Gerken, G; Canbay, A: Long-term maintenance of human liver tissue by ex-vivo perfusion of liver sections
P_3_20
Holzer, K; Ori, A; Winkler, J; Eiteneuer, E; Breuhahn, K; Beck, M; Schirmacher, P; Singer, S: Nup155 is linked to the p53 pathway in HCC
P_3_21
Stremmel, W; Staffer, S; Wannhoff, A; Pathil-Warth, A; Chamulitrat, W: Phospholipase A2 controls fatty acid uptake into liver cells in a pharmacologically-responsive way
P_3_23
Kaldenbach, M; Erschfeld, S; Cubero, J; Fragoulis, A; Schumacher, F; Kroy, D; Wruck, C; Trautwein, C; Streetz, K: Protektive Wirkung des HGF/c-Met bei Acetaminophen induziertem Leberschaden durch Inhibierung von oxidativem Stress
P_3_25
Straub, BK; Pawella, LM; Hashani, M; Eiteneuer, E; Schirmacher, P: Sequential PAT-expression in human hepatocyte steatosis
P_3_28
Marbach, E; Matz-Soja, M; Klöting, N; Kern, M; Gebhardt, R: TICK-TOCK-HEDGEHOG: Hedgehog signalling controls the liver circadian clock and vice versa
P_3_30
Böttger, J; Günter, K; Aleithe, S; Matz-Soja, M; Marbach, E; Hofmann, U; Gebhardt, R: Wnt-signaling gradients and their impact on liver parenchymal heterogeneity – a link to xenobiotic metabolism
P_3_31
Drescher, HK; Schippers, A; Erschfeld, S; Noels, H; Wagner, N; Trautwein, C; Streetz, KL; Kroy, DC: β7-Integrins and MAdCAM-1 play an opposing role during the development of Non-alcoholic steatohepatitis (NASH)