Int J Sports Med 2008; 29(2): 116-119
DOI: 10.1055/s-2007-965819
Physiology & Biochemistry

© Georg Thieme Verlag KG Stuttgart · New York

Cardiovascular Effects of Cadence and Workload

J. L. Moore1 , J. D. Shaffrath2 , G. A. Casazza3 , C. L. Stebbins4
  • 1Sports Medicine, University of California, Davis, Sacramento, California, United States
  • 2Neurobiology, Physiology, and Behavior, University of California, Davis, Davis, California, United States
  • 3Sports Medicine, University of California, Davis, Davis, California, United States
  • 4Internal Medicine, Division of Cardiovascular Medicine, University of California, Davis, Davis, California, United States
Further Information

Publication History

accepted after revision July 13, 2007

Publication Date:
24 October 2007 (online)

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Abstract

Increases in cadence may augment SV during submaximal cycling (> 65 % V·O2max) via effects of increased muscle pump activity on preload. At lower workloads (45 - 65 % V·O2max), SV tends to plateau, suggesting that effects of increases in cadence on pump activity have little influence on SV. We hypothesized that cadence-induced increases in CO at submaximal workloads, where SV tends to plateau, are due to elevations in HR and/or O2 extraction. SV, CO, HR, V·O2, and Δa - vO2 were assessed at 80 and 100 rpm during workloads of 50 % (LO) or 65 % (HI) of V·O2max in 11 male cyclists. No changes in SV were seen. CO was higher at 100 rpm in 10 of 11 subjects at LO (18.1 ± 2.7 vs. 17.2 ± 2.6 L/min). V·O2 at both workloads was greater at 100 than 80 rpm as was HR (LO: 129 ± 11 vs. 121 ± 10 beats/min; HI: 146 ± 13 vs. 139 ± 14 beats/min) (p < 0.05). Δa - vO2 was greater at HI compared to LO at 80 (15.1 ± 1.6 vs. 13.6 ± 1.3 ml) and 100 rpm (16.0 ± 1.7 vs. 15.1 ± 1.6 ml) (p < 0.05). Results suggest that increases in O2 demand during low submaximal cycling (50 % V·O2max) at high cadences are met by HR-induced increases in CO. At higher workloads (65 % V·O2max), inability of higher cadences to increase CO and O2 delivery is offset by greater O2 extraction.

References

Dr. Ph.D. Charles L. Stebbins

Internal Medicine, Division of Cardiovascular Medicine
University of California, Davis

One Shields Ave. TB - 172

95616 Davis, California

United States

Email: clstebbins@ucdavis.edu