Semin Liver Dis 2023; 43(03): e2
DOI: 10.1055/s-0043-1776036
Erratum

Erratum to: Genetic Contributions to Biliary Atresia: A Developmental Cholangiopathy

Authors

  • Dominick J. Hellen

    1   Division of Pediatric Gastroenterology, Hepatology, and Nutrition, Children's Healthcare of Atlanta and Emory University School of Medicine, Atlanta, Georgia
  • Saul J. Karpen

    1   Division of Pediatric Gastroenterology, Hepatology, and Nutrition, Children's Healthcare of Atlanta and Emory University School of Medicine, Atlanta, Georgia
Preview

The authors have informed the publisher that in this article (with DOI 10.1055/a-2153-8927; published online on September 19, 2023), [Tables 3] and [4] have been swapped. The correct [Table 3] and [Table 4] are as follows:

Table 3

Biliary atresia-associated microRNAs and their hypothesized target genes

miRNA

Target

Analyses

miR-19b

TGFßR2

140

miR-21

RASGRP1 (upstream of Erk/Mek), PTEN

141,142

miR-29b,c

DNMT3A, DNMT3B, IFN-γ

115,136,143

miR-140–3p

FGF9

144

miR-142–5p

IFN-γ

136

miR-145

ADD3

37

miR-155

SOCS1, IFN-γ

145,146

miR-200b

FOG2

116,144

miR-214

SUFU

147

miR-499

SOX6, ETS1

148,149

miR-4429

MAPK1, AKT3, PTEN

150

miR-4689

SGK1

150

Table 4

Hypomethylated regions identified in biliary atresia patients

Hypomethylated gene (or region)

Cell type

Analyses

SHH, GLI2, PDGFA

Hepatocytes, cholangiocytes

101

Alu, LINE-1, SAT2 (retrotransposons)

Peripheral blood leukocytes

118

IFN-γ promoter

Hepatocytes, cholangiocytes, CD4+ T cells

136,137

ATX promoter

Peripheral blood leukocytes

138

CD11a promoter

CD4+ T cells

120

FOXP3 promoter

Treg cells

139



Publikationsverlauf

Artikel online veröffentlicht:
12. Oktober 2023

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