J Pediatr Intensive Care 2018; 07(01): 046-048
DOI: 10.1055/s-0037-1602803
Case Report
Georg Thieme Verlag KG Stuttgart · New York

Reversible Dilated Cardiomyopathy Due to Combination of Vitamin D–Deficient Rickets and Primary Hypomagnesemia in an 11-Month-Old Infant

Mutlu Uysal Yazici
1  Department of Pediatric Intensive Care, Hacettepe University, Ankara, Turkey
,
Selman Kesici
1  Department of Pediatric Intensive Care, Hacettepe University, Ankara, Turkey
,
Hüseyin Demirbilek
2  Department of Pediatric Endocrinology and Metabolism, Hacettepe University, Ankara, Turkey
,
Murat Tanyıldız
1  Department of Pediatric Intensive Care, Hacettepe University, Ankara, Turkey
,
Mehmet Gumustas
3  Department of Pediatric Cardiology, Hacettepe University, Ankara, Turkey
,
Benan Bayrakci
1  Department of Pediatric Intensive Care, Hacettepe University, Ankara, Turkey
› Author Affiliations
Funding None.
Further Information

Publication History

25 December 2016

29 March 2017

Publication Date:
02 May 2017 (eFirst)

Abstract

Vitamin D–deficient rickets is still an important and common health problem in developing countries. Since calcium is an essential ion for cardiac muscle contraction, calcium deficiency caused by rickets can cause secondary dilated cardiomyopathy. This situation can be exacerbated by coexisting hypomagnesemia. Here, we report a case of dilated cardiomyopathy due to hypocalcemia induced by vitamin D–deficient rickets and accompanying primary hypomagnesemia in an infant whose cardiomyopathy was successfully treated by replacement of calcium, vitamin D, and magnesium. In addition to genetic causes, viral infections, and idiopathic factors, metabolic abnormalities are important etiologic factors in pathogenesis of dilated cardiomyopathy and since they are treatable, prompt diagnosis of these disorders is crucial.

Note

This study was conducted at Hacettepe University, Faculty of Medicine, Pediatric Intensive Care Unit, Ankara, Turkey.