Platelets from a patient heterozygous for the defect of P2cyc receptors for ADP have a secretion defect despite normal thromboxane A2 production and normal granule stores: Further evidence that some cases of platelet ‘primary secretion defect’ are heterozygous for a defect of P2cyc receptors - Cattaneo, M; Lecchi, A; Lombardi, R (Arterioscler Thromb Vasc Biol, 2000)
et al.