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Dtsch Med Wochenschr 2006; 131(27): 1541-1545
DOI: 10.1055/s-2006-947793
DOI: 10.1055/s-2006-947793
Prinzip & Perspektive | Review article
Nephrologie, Molekularbiologie
© Georg Thieme Verlag KG Stuttgart · New York
Fokal-segmentale Glomerulosklerose (FSGS)
Molekulare Defekte und klinische RelevanzFocal segmental glomerulosclerosis (FSGS)Molecular defects and clinical relevanceFurther Information
Publication History
eingereicht: 18.1.2006
akzeptiert: 8.5.2006
Publication Date:
30 June 2006 (online)
Schlüsselwörter
fokal-segmentale
Glomerulosklerose - nephrotisches Syndrom - Podozyten
Keys words
focal segmental
glomerulosclerosis - nephrotic syndrome - podocytes
Literatur
- 1 D’Agati V D, Fogo A B, Bruijn J A, Jenette J C. Pathologic classification of focal segmental glomerulosclerosis: a working proposal. Am J Kidney Dis. 2004; 43 368-382
- 2 Massague J. How cells read TGF-beta signals. Nat Rev Mol Cell Biol. 2000; 1 169-178
- 3 Schiffer M, Bitzer M, Roberts I SD. et al . Apoptosis in podocytes induced by TGF-beta and Smad7. J Clin Invest. 2001; 108 807-816
- 4 Vincenti F, Ghiggeri G M. New insights into the pathogenesis and the therapy of recurrent focal glomerulosclerosis. Am J Transplant. 2005; 5 1179-1185
- 5 Wharram B L, Goyal M, Wiggins J E. et al . Podocyte depletion causes glomerulosclerosis: diphtheria toxin-induced podocyte depletion in rats expressing human diphtheria toxin receptor transgene. J Am Soc Nephrol. 2005; 16 2941-2952
Dr. med. Mario Schiffer
Nachwuchsgruppenleiter im Emmy Noether-Programm, Abteilung für Nieren- und Hochdruckkrankheiten, Medizinische Hochschule Hannover
Carl Neuberg Straße 1
30625 Hannover
Phone: 0511/5324708
Fax: 0511/552366
Email: schiffer.mario@mh-hannover.de