DOI:
10.1055/s-00000017
Experimental and Clinical Endocrinology & Diabetes
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References
Alarcon C, Leahy JL, Schuppin GT, Rhodes CJ.
Increased secretory demand rather than a defect in the proinsulin conversion mechanism causes hyperproinsulinemia in a glucose-infusion rat model of non-insulin-dependent diabetes mellitus.
J Clin Invest. 95: 1032-1039 1995;
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