Biomechanics in vascular biology and cardiovascular disease

Thromb Haemost 2016; 115(03): 465-466
DOI: 10.1160/th16-01-0075

Theme Issue Editorial

Schattauer GmbH

Paul C. Evans

¹Department of Cardiovascular Science, University of Sheffield, Sheffield, UK

,

Frank J. H. Gijsen

²Department of Biomedical Engineering, Thoraxcenter, Erasmus Medical Center, Rotterdam, the Netherlands

,

Jolanda J. Wentzel

²Department of Biomedical Engineering, Thoraxcenter, Erasmus Medical Center, Rotterdam, the Netherlands

,

Kim Van der Heiden

²Department of Biomedical Engineering, Thoraxcenter, Erasmus Medical Center, Rotterdam, the Netherlands

References
1 Lehoux S, Jones EA. Shear stress, arterial identity and atherosclerosis. Thromb Haemost 2016; 115: 467-473.
2 Maimari N, Pedrigi RM, Russo A. et al. Integration of flow studies for robust selection of mechano-responsive genes. Thromb Haemost 2016; 115: 474-483.
3 Morbiducci U, Kok AM, Kwak BR. et al. Atherosclerosis at arterial bifurcations: evidence for the role of haemodynamics and geometry. Thromb Haemost 2016; 115: 484-492.
4 Speelman L, Teng Z, Nederveen AJ. et al. MRI based biomechanical parameters for carotid artery plaque vulnerability assessment. Thromb Haemost 2016; 115: 493-500.
5 Van der Heiden K, Hoogendoorn A, Daemen MJ. et al. Animal models for plaque rupture: a bio-mechanical assessment. Thromb Haemost 2016; 115: 501-508.
6 White SJ, Newby AC, Johnson TW. White En-dothelial Erosion of Plaques as a substrate for coronary thrombosis. Thromb Haemost 2016; 115: 509-519.