XXVI. Jahrestagung der Kind-Philipp-Stiftung für Leukämieforschung

Abstracts (HTML) List of Authors

Samareh, B

Hematopoietic-specific lyn-substrate 1 (HCLS) is deacetylated by NAMPT/NAD+/SIRT1 pathway

Schewe, D

TGF-β signaling causes ALL quiescence
p38α/β inhibition prevents ALL proliferation in vivo

Schier, MC

GRHL1 inhibits tumorigenicity and is a prognostic marker in neuroblastoma

Schlegelberger, B

The Association of Mutations in RUNX1 and CSF3R with the Development of Leukemia in Severe Congenital Neutropenia: A unique pathway in leukemogenesis

Schmidt, F

Molecular characterization of lineage-specific long non-coding RNAs in hematopoiesis

Schmidt, R

A novel treatment stratification system for current low- and intermediate-risk neuroblastoma patients using gene expression-based classification

Schnittger, S

The Association of Mutations in RUNX1 and CSF3R with the Development of Leukemia in Severe Congenital Neutropenia: A unique pathway in leukemogenesis

Schomburg, A

Dysregulated expression of histone deacetylases in medulloblastoma – potential targets for selective inhibitor treatment

Schramm, A

Targeting the disialoganglioside GD2 in neuroblastoma using a phage display derived oligopeptide
NTRK1/TrkA expression is a potential regulator of neuroblastoma cross-talk with the schwannian stroma

Schrappe, M

Prediction of minimal residual disease in pediatric ALL: Evaluation of the predictive potential of the variables from the ALL-BFM 2000 trial
TGF-β signaling causes ALL quiescence
p38α/β inhibition prevents ALL proliferation in vivo
Identification of germline polymorphisms associated with clinical and toxicity phenotypes in childhood acute lymphoblastic leukemia

Schrauder, A

Prediction of minimal residual disease in pediatric ALL: Evaluation of the predictive potential of the variables from the ALL-BFM 2000 trial

Schulte, JH

NTRK1/TrkA expression is a potential regulator of neuroblastoma cross-talk with the schwannian stroma

Schweinitz, D von

Deciphering the genetic origin of childhood liver cancer

Seidel, D

Fenretinide (4-HPR) as immunostimulator of the antibody-dependent (ADCC) and antibody-independent NK cell mediated cytotoxicity (AICC) on neuroblastoma

Sherkheli, F

Functional characterization of somatically mutated genes in neuroblastoma
The transcription factor activating protein 2 beta (TFAP2B) mediates neuronal differentiation in neuroblastoma cells

Siebert, N

Genotyping of KIR/KIRL mismatch, FCGR2A, -3A and -3B polymorphisms in patients subject to high-risk neuroblastoma immunotherapy using melting curve analysis
Development of dual function MYCN-DNA vaccines for anti-neuroblastoma immunotherapy
Long-term infusion of anti-GD2 antibody ch14.18/CHO is active and less toxic in high-risk neuroblastoma patients
Fenretinide (4-HPR) as immunostimulator of the antibody-dependent (ADCC) and antibody-independent NK cell mediated cytotoxicity (AICC) on neuroblastoma
Generation and characterization of ganglidiximab for active immunotherapy of neuroblastoma

Skokowa, J

Hematopoietic-specific lyn-substrate 1 (HCLS) is deacetylated by NAMPT/NAD+/SIRT1 pathway
The Association of Mutations in RUNX1 and CSF3R with the Development of Leukemia in Severe Congenital Neutropenia: A unique pathway in leukemogenesis
Identification of new leukemia associated gene mutations by whole genome sequencing of DNA from two siblings with congenital neutropenia and secondary AML
Acetylation of LEF-1 transcription factor regulating hematopoietic differentiation

Stachorski, L

Characterization of oncogenes on chromosome 21 identified by shRNA-based viability screening

Stanulla, M

Prediction of minimal residual disease in pediatric ALL: Evaluation of the predictive potential of the variables from the ALL-BFM 2000 trial
The Association of Mutations in RUNX1 and CSF3R with the Development of Leukemia in Severe Congenital Neutropenia: A unique pathway in leukemogenesis
TGF-β signaling causes ALL quiescence
p38α/β inhibition prevents ALL proliferation in vivo
Identification of germline polymorphisms associated with clinical and toxicity phenotypes in childhood acute lymphoblastic leukemia

Steinbronn, N

Continuous but not intermittent long-term exposure to the tyrosine kinase inhibitor (TKI) dasatinib causes cardiac failure in juvenile rats

Steinemann, D

The Association of Mutations in RUNX1 and CSF3R with the Development of Leukemia in Severe Congenital Neutropenia: A unique pathway in leukemogenesis

Stermann, A

Development of dual function MYCN-DNA vaccines for anti-neuroblastoma immunotherapy
Fenretinide (4-HPR) as immunostimulator of the antibody-dependent (ADCC) and antibody-independent NK cell mediated cytotoxicity (AICC) on neuroblastoma

Sterz, C

A novel treatment stratification system for current low- and intermediate-risk neuroblastoma patients using gene expression-based classification

Strasser, RH

Continuous but not intermittent long-term exposure to the tyrosine kinase inhibitor (TKI) dasatinib causes cardiac failure in juvenile rats

Strom, TM

Deciphering the genetic origin of childhood liver cancer

Strube, S

TGF-β signaling causes ALL quiescence
p38α/β inhibition prevents ALL proliferation in vivo

Süss, R

Imaged-guided Therapy of Ewing Sarcoma Using Magnetic Nanoparticles Coupled with anti-CD99-Antibody and siRNA against EWS/FLI1

Suttorp, M

Continuous but not intermittent long-term exposure to the tyrosine kinase inhibitor (TKI) dasatinib causes cardiac failure in juvenile rats
Long-term administration of the tyrosine kinase inhibitor bosutinib via micro-osmotic pumps and resulting skeletal side effects in juvenile rats