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Thromb Haemost 1999; 81(02): 322-323
DOI: 10.1055/s-0037-1614469
DOI: 10.1055/s-0037-1614469
Letters to the Editor
Assessment of the Expression of Candidate Human Plasma Membrane Phospholipid Scramblase in Scott Syndrome Cells
Further Information
Publication History
Received15 September 1998
Accepted26 October 1998
Publication Date:
08 December 2017 (online)
* CRTH-CHRU, Nantes, France
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References
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- 3 Toti F, Satta N, Fressinaud E, Meyer D, Freyssinet JM. Scott syndrome, characterized by impaired transmembrane migration of procoagulant phosphatidylserine and hemorrhagic complications, is an inherited disorder. Blood 1996; 87: 1409-15.
- 4 Zhou Q, Zhao J, Stout JG, Luhm RA, Wiedmer T, Sims PJ. Molecular cloning of human plasma membrane phospholipid scramblase. J Biol Chem 1997; 272: 18240-4.
- 5 Zhou Q, Sims PJ, Wiedmer T. Expression of proteins controlling transbilayer movement of plasma membrane phospholipids in the B lymphocytes from a patient with Scott syndrome. Blood 1998; 92: 1707-12.
- 6 Zhao J, Zhou Q, Wiedmer T, Sims PJ. Level of expression of phospholipid scramblase regulates induced movement of phosphatidylserine to the cell surface. J Biol Chem 1998; 273: 6603-6.
- 7 Zhou Q, Sims PJ, Wiedmer T. Identity of a conserved motif phospholipid scramblase that is required for Ca2+-accelerated transbilayer movement of membrane phospholipids. Biochemistry 1998; 37: 2356-60.
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