Int J Sports Med
DOI: 10.1055/s-0034-1395508
Physiology & Biochemistry
© Georg Thieme Verlag KG Stuttgart · New York

Effects of Walking with Blood Flow Restriction on Excess Post-exercise Oxygen Consumption

G. V. Mendonca
Laboratory of Motor Behavior, Faculdade de Motricidade Humana, Universidade de Lisboa, Portugal
,
J. R. Vaz
Laboratory of Motor Behavior, Faculdade de Motricidade Humana, Universidade de Lisboa, Portugal
,
P. Pezarat-Correia
Laboratory of Motor Behavior, CIPER, Faculdade de Motricidade Humana, Universidade de Lisboa, Portugal
,
B. Fernhall
Kinesiology, Nutrition, and Rehabilitation, University of Illinois at Chicago, United States
› Author Affiliations
Further Information

Publication History



accepted after revision 10 September 2014

Publication Date:
09 February 2015 (eFirst)

Abstract

This study determined the influence of walking with blood flow restriction (BFR) on the excess post-exercise oxygen consumption (EPOC) of healthy young men. 17 healthy young men (22.1±2.9 years) performed graded treadmill exercise to assess VO2peak. In a randomized fashion, each participant performed 5 sets of 3-min treadmill exercise at their optimal walking speed with 1-min interval either with or without BFR. Participants were then seated in a chair and remained there for 30 min of recovery. Expired gases were continuously monitored during exercise and recovery. BFR increased the O2 cost of walking as well as its relative intensity and cumulative O2 deficit (p<0.05). The EPOC magnitude after walking with BFR was greater than in the non-BFR condition (p<0.05). No differences between conditions were seen for the duration of EPOC. The EPOC magnitude was no longer different between conditions after controlling for the differences in relative intensity and in the cumulative O2 deficit (p>0.05). These data indicate that walking with BFR increases the magnitude of EPOC. Moreover, they also demonstrate that such increment in EPOC is likely explained by the effects of BFR on walking relative intensity and cumulative O2 deficit.