ABSTRACT
Which site(s) of brain damage are associated with apraxia of speech (AOS)? There appears
to be little agreement. The article first considers some reasons why not. Even allowing
for factors that may have influenced findings, a definitive answer to the question
of the neurological bases of AOS is not currently possible. The article goes on to
look briefly at developments in the field of motor control, and limb and buccofacial
apraxia in particular, that may hold clues to an answer or at least to asking the
right questions. In particular, if AOS is to be understood as a motor disorder, then
models compatible with motor control and its neurophysiological underpinnings must
be sought. Current models of motor control and apraxia stress the sensorimotor, distributed,
interactive nature of control across multiple brain areas.
KEYWORDS
Apraxia of speech - motor control - limb apraxia